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This technique leverages the reversible self-assembly properties of viral capsid proteins, which can be dismantled under controlled environmental stressors–such as high salt concentrations, chelators, or reducing agents–and subsequently triggered to reform in the presence of target cargo (Figure 3A

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This last possibility acquires greater relevance if we take into account the fact that the only currently available strategy is the use of immunosuppresors which increase the incidence of tumors and infectious bacterial, fungal and viral diseases in patients with transplants (73)